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May 26Liked by Will Falconer, DVM

RIP Dr Ronald Schultz ❤️

Immunological memory is the ability of the immune system to quickly and specifically recognize an antigen that the body has previously encountered and initiate a corresponding immune response. Generally these are secondary, tertiary and other subsequent immune responses to the same antigen. Immunological memory is responsible for the adaptive component of the immune system, special T and B cells — the so-called memory T and B cells.

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IgG antibodies are Y-shaped protein molecules that are produced by special white blood cells (B lymphocytes) in response to foreign substances (antigens) such as viruses or bacteria. Antibodies can attach to these viruses or bacteria, rendering them harmless and unable to penetrate healthy cells. They GO AWAY with time and could fall to a NEGATIVE level DOESN'T mean susceptible.

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Total immunity against viral diseases includes:

1. Local IgA and IgM

2. Humoral immunity of IgG antibodies, both those present in the blood AND those that can be produced quickly when the antigen is present

3. Cellular immunity or MEMORY

4. Other mechanisms.

When we measure antibody titers, we are ONLY documenting the IgG antibodies present in the bloodstream.

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The Immune System fires up when a pathogen, like a virus, enters the body. The pathogen releases a protein called an antigen, which calls into action the immune system’s special disease-fighting cells. "Called B and T lymphocytes", these cells NOT only destroy the virus, but they REMEMBER what it looked like so they can fend it off in the FUTURE.

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IMMUNOLOGIC MEMORY allows the immune system to REMEMBER the antigens or organisms to which it has previously been exposed. MEMORY EFFECTOR B cells (long-lived plasma cells) and MEMORY T-cells specific to a virus, give long-term immunity against these diseases.

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ADAPTIVE IMMUNITY.

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The “Adaptive Immune Response" is younger than the “Innate Immune Response" in evolutionary terms and is more specific and considerably MORE POTENT in its effects.

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The constituents of the adaptive immune response are the lymphoid cells. These include:

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The T lymphocytes and the cytokine and chemokine messenger proteins released by these cells, which direct and REGULATE the adaptive immune response.

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The B lymphocytes, which transform to the late-stage plasma cells that produce and secrete antibody.

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The lymphoid cells of the adaptive immune response reside in, and circulate between, the various lymphoid tissues of the body (e.g. the lymph nodes, spleen and mucosal lymphoid tissues). In the adaptive immune response, antigen is first transported from a site of infection by a dendritic cell to the regional lymphoid tissue. That dendritic cell in turn activates "Antigen-Specific T lymphocytes," which further activate Antigen-Specific B lymphocytes.”

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These activated, Antigen-Specific lymphocytes must then be mobilized from the regional lymphoid tissue and sent to the site of infection, a process that involves these cells moving into the lymphatic and blood circulation and interacting with the endothelial lining of blood vessels.

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Once these cells reach the site of infection, they are able to mount a full-scale ‘EFFECTOR’ response, which is considerably STRONGER than that permitted by innate immunity. As these processes take some time to occur (in the order of 4–7 days), there is a delay before adaptive immunity ‘takes over’ from the innate form of defence.

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The final component of Adaptive Immunity is the development of a “REGULATORY RESPONSE" that will "SWITCH OFF" the system when it is NO LONGER REQUIRED (i.e. when the pathogen has been ELIMINATED) so as NOT to cause DAMAGE to normal body tissue.

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HOWEVER, once this is achieved, the immune system retains the “MEMORY” of that immune response.

IMMUNOLOGICAL MEMORY is another key feature of the Adaptive Immune Response. MEMORY allows the generation of a much MORE effective SECONDARY IMMUNE RESPONSE (Anamnestic MEMORY Response) if that same antigen is EVER RE-ENCOUNTERED.

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author

Absolutely correct and thanks for the details. I wrote about this in The Fallacy of Titer Tests: http://vitalanimal.com/fallacy-of-titer-tests/

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May 26Liked by Will Falconer, DVM

I know and I've shared it several times in the past, thank you for everything, I did my version from learning from people like you and many others and Dr Ronald Schultz videos and his words..

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May 26Liked by Will Falconer, DVM

Thank you very much, great information

When I am ready for my next dog he/she will benefit greatly

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May 27Liked by Will Falconer, DVM

Could you use raw goat milk to make kefir and give that to dog?

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author

By all means, yes.

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Thank you!!

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May 26Liked by Will Falconer, DVM

Great stack Dr. Will. Thank you ❤️

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I love your stack, your observations, your knowledge, and your compassion. I believe you walk in the footsteps of St. Francis. You're doing very important work on behalf of animals and I thank you.

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We give our dogs raw milk

We had someone telm us we needed to feed them

Propylene glycol and cooked white potatoes and carrots🤔

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author

Wow. The two almost couldn't be further apart.

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Just got a puppy who had 5-1 vacc at six weeks and 5-1 vacc at eight weeks. Was planning on doing the parvo and distemper protocol and received your nosodes. But after getting the puppy realized they lied to me about his vacc. Can I still do your parvo and distemper protocol even though he has had the shots. Have not taken him to the vet here and done any tigers.

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author

Lied in what way?

And: you can always do nosodes, right up till 9 mo of age. https://vitalanimal.com/37-nosode-safe-after-vaccines/

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May 27Liked by Will Falconer, DVM

They told me they did not do 5-1 vacs, when I got the vac report it showed the 5-1 at six weeks and eight weeks. This puppy will max out at only 18-20 pounds😔. My brother brought the puppy to me from out of state. I did your Nux detox already but honestly, I was nauseous when I read what he had. 2 wks Deworm Pyrantel, 3 wks same, 4 wks Fenbendazole, 5 wks same. TruCan DAPPI+C contains Canine distemper, Adenovirus Type 2, Coronavirus, Parinfluenza & parvo, Nobivac Intra-Trac 3 contains Canine Adenovirus type 2, Parinfluenza, Bordetella. 8 weeks TruCan DAPPI+CL4 contains Canine Distrmper, Adenovirus Type 2 coronavirus, Parainfluenza, Parvo,& Leptospira Canicola-Grippotyphosa Icterohaemorrhagiar- Pomona Bacterin. 48 hours prior to leaving Drontal Plue - Intestinal Parasite Prevention & Capstar - Flea Prevention. How is this dog still alive? Thank you thank you for the article I can still follow thru with the nosodes. He is currently 14 weeks. He was transitioned to raw feed 1 week after I got him. The only thing I could detect was he had I believe Picca, ate twigs, leaves, dirt pulled grass up by the roots and ate it. Almost non existent at this point.

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Honestly, I’ve been using homeopathy for the last 5 years (I wish it was more like 25 years)😉 Just was really nervous about using the nosodes after all the vaccs.

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May 26·edited May 26

MDA can be present up to 25 weeks every pup is an individual, if breeders are going to do a jab they should do what is done in the Netherlands, titer pups to watch MDA going down till it’s at S1/0 before injecting. Then titer again 3-4 weeks later to see if it’s worked.. Aim is one for life.. Chances are them injections were useless..They do a lot of vaccinating but not a lot of immunising…Dr Ronald Schultz..

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Thank you for your reply

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deletedMay 26
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A worthy question, and it makes me think of Sarah, a long ago client who took my teaching deeply and vowed she'd not take anything from the vets other than what she came for, which was an annual heartworm test.

The clinic got to know her signature big grin and concurrent refusal to update her long "out of date" vaccines with her foursome of dogs. They stopped asking, for the most part, after hearing her kind refusal (amid a big smile): "No, we're only here for the HW test."

Each generation of her favorite Akitas got healthier from her temerity and good work, largely refusing (with a smile, always) to take the "prevention" she knew to be dangerous. No fear, no enmity, just determination and understanding that she, alone, was the one "calling the shots" for her pack. (And, of course, she also knew she'd be the one holding the bag if they damaged her dogs).

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